Publication date: Jul 10, 2023
Granulocytes are innate immune effector cells with essential functions in host resistance to bacterial infections. I will discuss emerging evidence that during Mycobacterium tuberculosis infection, counter-intuitively, eosinophils are host-protective while neutrophils are host detrimental. Additionally, I will propose a ‘tipping-point’ model in which neutrophils are an integral part of a feedforward loop driving tuberculosis disease exacerbation.
Semantics
| Type | Source | Name |
|---|---|---|
| disease | MESH | disease exacerbation |
| disease | MESH | bacterial infections |
| disease | MESH | Mycobacterium tuberculosis infection |
| pathway | KEGG | Tuberculosis |
Original Article
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