Improvements in clinical signs and symptoms of Parkinson’s disease using photobiomodulation: a five-year follow-up.

Improvements in clinical signs and symptoms of Parkinson’s disease using photobiomodulation: a five-year follow-up.

Publication date: Oct 09, 2024

Parkinson’s disease is a progressive neurodegenerative disease characterized by clinical motor signs and non-motor symptoms that severely impact quality of life. There is an urgent need for therapies that might slow, halt or even reverse the progression of existing symptoms or delay the onset of new symptoms. Photobiomodulation is a therapy that has shown potential to alleviate some symptoms of Parkinson’s disease in animal studies and in small clinical trials. To assess long-term effectiveness of photobiomodulation therapy in a cohort of Parkinson’s disease individuals after five years of continuing therapy. Eight participants of the initial 12 in a previously published study agreed to be reassessed after five years. Seven of these participants had continued home-based, self-applied photobiomodulation therapy three times per week for five years. One participant had discontinued treatment after one year. Participants were assessed for a range of clinical motor signs, including MDS-UPDRS-III, measures of mobility and balance. Cognition was assessed objectively, and quality of life and sleep quality were assessed using self-reported questionnaires. A Wilcoxon Signed Ranks test was used to evaluate change in outcome measures between baseline (before treatment) and after five years, with the alpha value set to 0. 05. Of the seven participants who had continued photobiomodulation therapy, one had a preliminary diagnosis of multisystem atrophy and was excluded from the group analysis. For the remaining six participants, there was a significant improvement in walk speed, stride length, timed up-and-go tests, tests of dynamic balance, and cognition compared to baseline and nonsignificant improvements in all other measures, apart from MDS-UPDRS-III, which was unchanged and one measure of static balance (single leg stance, standing on the unaffected leg with eyes open) which declined. Five of six participants either improved or showed no decline in MDS-UPDRS-III score and most participants showed improvement or no decline in all other outcome measures. No adverse effects of the photobiomodulation therapy were reported. This study provides a signal that photobiomodulation therapy might safely reduce important clinical motor signs and non-motor symptoms in some Parkinson’s disease patients, with improvements maintained over several years. Home-based photobiomodulation therapy has the potential to complement standard therapies to manage symptoms and potentially delay Parkinson’s symptom progression. Australian New Zealand Clinical Trials Registry, registration number ACTRN12618000038291p, registered on 12/01/2018.

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Concepts Keywords
Actrn12618000038291p Aged
Australian Cognition
Parkinson Female
Photobiomodulation Follow-Up Studies
Week Humans
Low-Level Light Therapy
Male
Middle Aged
Mobility
Motor symptoms
Parkinson Disease
Parkinson’s disease
Photobiomodulation
Quality of Life
Sense of smell
Treatment Outcome

Semantics

Type Source Name
drug DRUGBANK Dopamine
disease MESH clinical progression
disease MESH gait
disease MESH death
pathway REACTOME Reproduction
disease MESH multisystem atrophy
disease MESH sleep quality
drug DRUGBANK Isoxaflutole
disease MESH Parkinson’s disease
drug DRUGBANK Methylergometrine
drug DRUGBANK ATP
drug DRUGBANK Oxygen
drug DRUGBANK Cyclic Adenosine Monophosphate
drug DRUGBANK Nitric Oxide
disease MESH osteoarthritis
disease MESH tendinopathies
disease MESH oral mucositis
disease MESH mucositis
disease MESH stroke
disease MESH post traumatic stress disorder
disease MESH depression
drug DRUGBANK Sodium lauryl sulfate
drug DRUGBANK Aspartame
disease MESH aids
drug DRUGBANK Water
drug DRUGBANK Medical air
drug DRUGBANK Cysteamine
drug DRUGBANK Coenzyme M
disease MESH respiratory tract infections
disease MESH rheumatoid arthritis
pathway KEGG Rheumatoid arthritis
drug DRUGBANK Levodopa
disease MESH arthritis
drug DRUGBANK Trestolone
disease MESH tremor
disease MESH dyskinesia
disease MESH back pain
disease MESH dementia
disease MESH double vision
disease MESH seizures
drug DRUGBANK Gold
drug DRUGBANK Carbidopa
disease MESH minimal clinically important difference
drug DRUGBANK Dimercaprol
disease MESH Cognitive impairment
drug DRUGBANK Methionine
disease MESH morbidities
disease MESH anosmia
disease MESH COVID 19
disease MESH neurodegenerative disorder
disease MESH Movement Disorder
disease MESH neurological disorders
disease MESH pathogenesis
pathway KEGG Parkinson disease
disease MESH phototoxicity
disease MESH Cancer
disease MESH brain disorders
disease MESH anxiety
disease MESH Alzheimer’s disease
drug DRUGBANK Trihexyphenidyl
drug DRUGBANK Profenamine
drug DRUGBANK Pentaerythritol tetranitrate
disease MESH anxiety disorders
disease MESH Parkinsonism
disease MESH Parkinson disease 8
drug DRUGBANK Elm
disease MESH daytime somnolence
pathway REACTOME Neurodegenerative Diseases
disease MESH Olfactory Impairment
disease MESH Ageusia

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