Modulation of RAAS receptors and miRNAs in COVID-19: implications for disease severity, immune response, and potential therapeutic targets.

Publication date: Mar 24, 2025

The SARS-CoV-2 spike protein interacts with ACE2, a key receptor within the renin-angiotensin-aldosterone system (RAAS), which plays a critical role in maintaining vascular homeostasis, regulating blood pressure, and modulating inflammation. An observational study analyzed the gene expression profiles of RAAS receptors and associated miRNAs in 88 hospitalized COVID-19 patients and 20 healthy controls, comparing the acute and post-acute phases to assess their impact on disease severity and recovery. Our findings revealed an association between reduced MAS1 expression in both advanced age (P = 0. 03) and the need for oxygen supplementation (P = 0. 04). Additionally, reduced ACE expression was associated with worse mortality outcomes (P = 0. 01). Notably, ACE2 and TMPRSS2 expression was significantly decreased (P 

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Concepts Keywords
Aldosterone ACE2
Homeostasis ACE2 protein, human
Mortality Adult
Proteins Aged
Therapeutic Angiotensin-Converting Enzyme 2
Angiotensin-Converting Enzyme 2
COVID-19
COVID-19
Female
Humans
Male
MAS1 protein, human
MicroRNAs
MicroRNAs
Middle Aged
miRNA
Proto-Oncogene Mas
Proto-Oncogene Mas
RAAS
Renin-Angiotensin System
SARS-CoV-2
SARS-CoV-2
Serine Endopeptidases
Serine Endopeptidases
TMPRSS2 protein, human

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