The synthetic lethal interaction between CDS1 and CDS2 is a vulnerability in uveal melanoma and across multiple tumor types.

Publication date: Jul 04, 2025

Metastatic uveal melanoma is an aggressive disease with limited effective therapeutic options. To comprehensively map monogenic and digenic dependencies, we performed CRISPR-Cas9 screening in ten extensively profiled human uveal melanoma cell line models. Analysis involved genome-wide single-gene and combinatorial paired-gene CRISPR libraries. Among our 76 uveal melanoma-specific essential genes and 105 synthetic lethal gene pairs, we identified and validated the CDP-diacylglycerol synthase 2 gene (CDS2) as a genetic dependency in the context of low CDP-diacylglycerol synthase 1 gene (CDS1) expression. We further demonstrate that CDS1/CDS2 forms a synthetic lethal interaction in vivo and reveal that CDS2 knockout results in the disruption of phosphoinositide synthesis and increased cellular apoptosis and that re-expression of CDS1 rescues this cell fitness defect. We extend our analysis using pan-cancer data, confirming increased CDS2 essentiality in diverse tumor types with low CDS1 expression. Thus, the CDS1/CDS2 axis is a therapeutic target across a range of cancers.

Concepts Keywords
Cancer Cds1
Crispr Cds2
Diacylglycerol Crispr
Knockout Diacylglycerol
Libraries Expression
Gene
Interaction
Lethal
Low
Melanoma
Synthase
Synthetic
Therapeutic
Tumor
Uveal

Semantics

Type Source Name
disease MESH uveal melanoma
disease MESH tumor
drug DRUGBANK Pentaerythritol tetranitrate
pathway REACTOME Apoptosis

Original Article

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