Protein discovery could reverse Huntington’s disease damage, study finds

Protein discovery could reverse Huntington’s disease damage, study finds

Publication date: Jul 07, 2025

(CREDIT: Nature Cell Death & Disease)Cell Traffic and Dead EndsIn 2014, the same team at UB discovered that the normal HTT protein worked like a traffic controller. (CREDIT: Nature Cell Death & Disease)Zooming in on the Cellular MapTo dig deeper, the team used stem cell-derived neurons from people with and without the HTT mutation. For years, researchers knew that HTT helped with moving cell parts along thin structures called axons, which are like highways inside neurons. (CREDIT: Nature Cell Death & Disease)Among the biggest disruptions was in a group of pathways related to axon guidance, membrane trafficking, and vesicle transport. It was actively interfering with other essential cell processes. They also saw higher levels of GSK3 and lower levels of ERK1 in the diseased neurons. These include the motor proteins that move cargo and the membrane proteins that receive signals from the rest of the body. In healthy cells, HTT helps bring other proteins together at membranes, like parts on a workbench.

Concepts Keywords
Buffalo Cells
Cancer Credit
Dead Damage
Healthier Death
Trafficking Disease
Erk1
Gsk3
Htt
Huntington
Huntingtons
Key
Mutation
Nature
Neurons
Transport

Semantics

Type Source Name
disease MESH death
drug DRUGBANK Guanine
drug DRUGBANK Adenine
drug DRUGBANK Pentaerythritol tetranitrate
disease MESH causes
disease MESH Huntington’s disease
pathway KEGG Motor proteins
pathway REACTOME Kinesins
drug DRUGBANK Phosphate ion
drug DRUGBANK Tropicamide
disease MESH defects
pathway REACTOME Axon guidance
pathway REACTOME Membrane Trafficking
disease MESH cancer
disease MESH Neurological Disorders
disease MESH Stroke
drug DRUGBANK Nonoxynol-9

Original Article

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