Rotarod training reverses corticosterone-induced motor deficits via oligodendrocyte lineage cell modulation.

Publication date: Jul 05, 2025

Adult-born oligodendrocytes are continuously produced in the brains of rodents. The functional role of these cells has been linked to the motor-related activities of healthy animals and is vital for acquiring new motor skills. However, the relationship between these cells and the control of motor-related activities has not been investigated in pathological conditions. Therefore, the aim of this study is to investigate the role of oligodendrocytes in depression-related motor deficits and the effects of training. Psychomotor retardation is a key symptom of depression. Consistent with the impairments observed in rodent motor performance, the proliferation and activation of adult-born oligodendrocytes are altered in a corticosterone-induced stress paradigm. Therapeutic rotarod training can alleviate these symptoms by reversing the aforementioned changes. Notably, these alterations are particularly pronounced in layer I of the motor cortex. Thus, this study provides evidence of the potential functional involvement of adult-born oligodendrocytes in the motor impairments observed in the depressed animals. Additionally, it offers preliminary results for further investigation into layer I of the motor cortex in relation to these pathological conditions.

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Concepts Keywords
Adult corticosterone-induced stress
Drugs exercise
Oligodendrocytes major depressive disorder
Rodents motor cortex
Training motor deficits
motor training
myelination
oligodendrocyte lineage cells
oligodendrocyte precursor cells
psychomotor retardation

Semantics

Type Source Name
drug DRUGBANK Corticosterone
disease MESH depression
disease MESH major depressive disorder

Original Article

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